Which 'Star Trek' crew wins out -- TV or movie?

By Randee Dawn, NBC News contributor

Casting any movie is tough, but try casting a "Star Trek" movie, knowing that the original television characters are already dearly beloved by millions of devoted fans. Few actors are as associated with their roles as William Shatner as Captain Kirk and Leonard Nimoy as Spock. Yet when Chris Pine and Zachary Quinto took over those roles in the 2009 film "Star Trek," reviews were mostly quite positive. As Pine, Quinto and co-stars prepare to beam down to theaters once again in "Star Trek Into Darkness," let's take a look at how the new actors fill out their Starfleet uniforms.

James T. Kirk
Classic:? William Shatner
New: Chris Pine

Paramount via Getty / Warner Bros

Chris Pine plays Capt. Kirk in the new "Star Trek" movies, but he can't beat the classic William Shatner.

The Shat has built a fine post-?Trek? career -- once he accepted with good humor that he would never, ever be forgotten as Kirk. But that aside, Classic Kirk has to rule over New Kirk. Classic Kirk had a twinkle in his eye, was never afraid to bare a chest, and had a way with a universe?s worth of ladies. We haven?t seen that yet in New Kirk, who?s more of a tomcat than a ladies? man and far from an unquestioned leader. Plus, only Classic Kirk has proven he can create a bazooka out of a log, dirt and gemstones.

Advantage: Classic Kirk

?

Leonard 'Bones' McCoy
Classic: DeForest Kelley
New: Karl Urban

Paramount via Getty / Warner Bros

Karl Urban and DeForest Kelley both have their good points as Bones, but the new guy, Urban, is a handsome bad boy.

Okay, so Classic Bones wasn?t afraid to smack a hoity-toity pregnant lady if he needed to do an examination. But something about his eternal grumpiness and pointed Spock-like eyebrows made Kelley's version of the doctor a little hard to like. New Bones is good looking enough to give Kirk a run for his money (if you like ?em dark and mercurial), he rocks a beard and swigs from a flask when necessary. New Bones is a bad boy as well as a doc, and that is the wave of the future.

Advantage: New McCoy

?

Spock
Classic: Leonard Nimoy
New: Zachary Quinto

Paramount via Getty / Warner Bro

Bravo, Zachary Quinto! The actor has taken on Leonard Nimoy's famed Spock and given him a modern update.

The old neck-pincher is one of the toughest decisions to make, particularly since Classic Spock (or rather, Spock Prime) makes an appearance in the rebooted series. The casting of Quinto as a youthful Nimoy is spot-on, unlike many of the other cast choices ? and the 2009 film even showed a pre-adolescent Spock on top of everything else. In a way, this is the best blended character with some of the series? most memorable personality quirks and special powers. How can we choose just one?

Advantage: Both Spocks

?

Montgomery 'Scotty' Scott
Classic: James Doohan
New: Simon Pegg

Paramount via Getty / Warner Bros

Simon Pegg isn't quite as devoted to the Enterprise as the original Scotty, James Doohan -- and that's a good thing.

You know, Classic Scotty really, really loved the Enterprise. Possibly a bit too much. Sure, he was the engineer and knew all of the inner workings of the ship, but sometimes you had to wonder if it made him a little touched in the head, being stuck down in the boiler room all the time. (No wonder he could outdrink an alien.) No such issue with New Scotty, who is still getting to know the big old girl and may take a long time to find his love connection. And that?s good for tension and comedy.

Advantage: New Scotty

?

Hikaru Sulu
Classic: George Takei
New: John Cho

Paramount via Getty / Warner Bros

Which Sulu rules, John Cho or George Takei? It's a tie!

In the 2009 film, Sulu got a few big moments ?- such as when he had to skydive from space and parachute onto a drilling platform, then fight off some Romulans. But the sword battle part of that scene was really an homage to one of Classic Sulu?s greatest scenes: When his inner swashbuckler comes to light and he runs around waving an epee. At the moment, there?s just not enough information on New Sulu to warrant leaving Classic Sulu behind, so we?d tend to lean toward Classic Sulu (oh, myyyy!) ? but let?s consider this the wild card in the bunch.

Advantage: Tie

?

Pavel Chekov
Classic: Walter Koenig
New: Anton Yelchin

Paramount via Getty / Warner Bros

New Chekov Anton Yelchin is a real Russian, unlike Walter Koenig from the classic series.

First off, if you?re gonna have a Russian on the Enterprise, hire a dang Russian. All due respect to Classic Chekov, but a Monkee look-alike from Chicago doesn?t hold much of a candle to New Chekov, whose portrayer hails from Leningrad and plays him as more delightfully impulsive than the originator. Plus, he was able to save Spock?s father thanks to some fantastical manipulation of the transporter. And he?s a navigator!

Advantage: New Chekov

?

Nyota Uhura
Classic: Nichelle Nichols
New: Zoe Saldana

Paramount via Getty / Warner Bros

Zoe Saldana's Uhura serves mostly as eye candy, whereas Nichelle Nichols juggled many roles.

Here?s a surprise: A show that began in the 1960s is better at handling its one regular female crew member than one in the new millennium. New Uhura served largely as eye candy and as a sex object for New Kirk, even if she could kick more butt and talk dirty. Classic Uhura managed to juggle all of her subspace frequencies far better -- and of course was the shared co-conspirator in one of TV?s first interracial kisses, when she and Classic Kirk locked lips.

Advantage: Classic Uhura

Final tallies: Four classics and five from the new bunch (though Spock and Sulu bridge both categories). Surprise! It turns out that the new franchise has managed to improve on the old -- though we sense there will be some disagreement on this issue. Tell us who your favorite newcomer is in our poll.

Source: http://entertainment.nbcnews.com/_news/2013/05/14/18254749-which-star-trek-crew-wins-out-tv-or-movie?lite

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Boosting 'cellular garbage disposal' can delay the aging process

May 6, 2013 ? UCLA life scientists have identified a gene previously implicated in Parkinson's disease that can delay the onset of aging and extend the healthy life span of fruit flies. The research, they say, could have important implications for aging and disease in humans.

The gene, called parkin, serves at least two vital functions: It marks damaged proteins so that cells can discard them before they become toxic, and it is believed to play a key role in the removal of damaged mitochondria from cells.

"Aging is a major risk factor for the development and progression of many neurodegenerative diseases," said David Walker, an associate professor of integrative biology and physiology at UCLA and senior author of the research. "We think that our findings shed light on the molecular mechanisms that connect these processes."

In the research, published today in the early online edition of the journal Proceedings of the National Academy of Sciences, Walker and his colleagues show that parkin can modulate the aging process in fruit flies, which typically live less than two months. The researchers increased parkin levels in the cells of the flies and found that this extended their life span by more than 25 percent, compared with a control group that did not receive additional parkin.

"In the control group, the flies are all dead by Day 50," Walker said. "In the group with parkin overexpressed, almost half of the population is still alive after 50 days. We have manipulated only one of their roughly 15,000 genes, and yet the consequences for the organism are profound."

"Just by increasing the levels of parkin, they live substantially longer while remaining healthy, active and fertile," said Anil Rana, a postdoctoral scholar in Walker's laboratory and lead author of the research. "That is what we want to achieve in aging research -- not only to increase their life span but to increase their health span as well."

Treatments to increase parkin expression may delay the onset and progression of Parkinson's disease and other age-related diseases, the biologists believe. (If parkin sounds related to Parkinson's, it is. While the vast majority of people with the disease get it in older age, some who are born with a mutation in the parkin gene develop early-onset, Parkinson's-like symptoms.)

"Our research may be telling us that parkin could be an important therapeutic target for neurodegenerative diseases and perhaps other diseases of aging," Walker said. "Instead of studying the diseases of aging one by one -- Parkinson's disease, Alzheimer's disease, cancer, stroke, cardiovascular disease, diabetes -- we believe it may be possible to intervene in the aging process and delay the onset of many of these diseases. We are not there yet, and it can, of course, take many years, but that is our goal."

'The garbage men in our cells go on strike'

To function properly, proteins must fold correctly, and they fold in complex ways. As we age, our cells accumulate damaged or misfolded proteins. When proteins fold incorrectly, the cellular machinery can sometimes repair them. When it cannot, parkin enables cells to discard the damaged proteins, said Walker, a member of UCLA's Molecular Biology Institute.

"If a protein is damaged beyond repair, the cell can recognize that and eliminate the protein before it becomes toxic," he said. "Think of it like a cellular garbage disposal. Parkin helps to mark damaged proteins for disposal. It's like parkin places a sticker on the damaged protein that says 'Degrade Me,' and then the cell gets rid of this protein. That process seems to decline with age. As we get older, the garbage men in our cells go on strike. Overexpressed parkin seems to tell them to get back to work."

Rana focused on the effects of increased parkin activity at the cellular and tissue levels. Do flies with increased parkin show fewer damaged proteins at an advanced age? "The remarkable finding is yes, indeed," Walker said.

Parkin has recently been shown to perform a similarly important function with regard to mitochondria, the tiny power generators in cells that control cell growth and tell cells when to live and die. Mitochandria become less efficient and less active as we age, and the loss of mitochondrial activity has been implicated in Alzheimer's, Parkinson's and other neurodegenerative diseases, as well as in the aging process, Walker said.

Parkin appears to degrade the damaged mitochondria, perhaps by marking or changing their outer membrane structure, in effect telling the cell, "This is damaged and potentially toxic. Get rid of it."

If parkin is good, is more parkin even better?

While the researchers found that increased parkin can extend the life of fruit flies, Rana also discovered that too much parkin can have the opposite effect -- it becomes toxic to the flies. When he quadrupled the normal amount of parkin, the fruit flies lived substantially longer, but when he increased the amount by a factor of 30, the flies died sooner.

"If you bombard the cell with too much parkin, it could start eliminating healthy proteins," Rana said.

In the lower doses, however, the scientists found no adverse effects. Walker believes the fruit fly is a good model for studying aging in humans -- who also have the parkin gene -- because scientists know all of the fruit fly's genes and can switch individual genes on and off.

Previous research has shown that fruit flies die sooner when you remove parkin, Walker noted.

Walker and Rana do not know what the optimal amount of parkin would be in humans.

While the biologists increased parkin activity in every cell in the fruit fly, Rana also conducted an experiment in which he increased parkin expression only in the nervous system. That, too, was sufficient to make the flies live longer.

"This tells us that parkin is neuroprotective during aging," Walker said. "However, the beneficial effects of parkin are greater -- twice as large -- when we increased its expression everywhere."

"We were excited about this research from the beginning but did not know then that the life span increase would be this impressive," Rana said.

The image that accompanies this news release shows clumps or aggregates of damaged proteins in an aged brain from a normal fly (left panel) and an age-matched brain with increased neuronal parkin levels (right panel). As can be seen, increasing parkin levels in the aging brain reduces the accumulation of aggregated proteins.

Scientists have found that this kind of protein aggregation occurs in mammals as well, including humans, Rana said.

"Imagine the damage the accumulation of protein trash is doing to the cell," Walker said. "With increased Parkin, the trash has been collected. Without it, the garbage that should be discarded is accumulating in the cells."

Disclaimer: This article is not intended to provide medical advice, diagnosis or treatment. Views expressed here do not necessarily reflect those of ScienceDaily or its staff.

Source: http://feeds.sciencedaily.com/~r/sciencedaily/top_news/~3/iQTWJf6iw4c/130506181619.htm

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Stars put punk edge in their Met Gala outfits

NEW YORK (AP) ? Beyonce made a grand entrance in a flame-motif Givenchy gown at the Met Gala, a big fundraiser for the Metropolitan Museum of Art.

There was a punk theme to Monday's event to embrace the featured exhibit inside called "Punk: Chaos to Couture."

Rooney Mara wore a white lace gown with zip front, escorted by Givenchy creative director Riccardo Tisci. She says she never went through a punk fashion phase but "I was definitely a punk."

Guests went with an edgy look. Miley Cyrus' sheared hair complemented a glittery net gown by Marc Jacobs.

Anne Hathaway, in her new short blond hairdo, wore burned-out vintage Valentino.

Sarah Jessica Parker wore a graffiti splashed and slashed gown by Giles Deacon with a feather mohawk, and Kim Kardashian wore a high-neck floral gown by Tisci.

Source: http://news.yahoo.com/stars-put-punk-edge-met-gala-outfits-003952514.html

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Health Book Review: Healing Hepatitis and Liver Disease Naturally ...

Visit www.HealthBookMix.com for more health and fitness book reviews! This is an audio summary of Healing Hepatitis and Liver Disease Naturally: Detoxification. Liver gall bladder flush & Cleanse. Cure Hepatitis C and Hepatitis B. Lower blood cholesterol and stop cirrhosis by Peter Oyakhire.

Source: http://juicefasting.fitnessthroughfasting.com/juice-fasting-detoxification/health-book-review-healing-hepatitis-and-liver-disease-naturally-detoxification-liver-gall-bladder-flush-cleanse-cure-hepatitis-c-and-hepatitis-b-lower-blood-cholesterol-and-stop-cirrhosis.php

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Don't txt n drive: Teens not getting msg: 43 percent of youths admit to texting while driving

May 4, 2013 ? Teens can get hundreds of text messages a day, but one message they aren't getting is that they shouldn't text and drive.

Nearly 43 percent of high school students of driving age who were surveyed in 2011 reported texting while driving at least once in the past 30 days, according to a study to be presented Saturday, May 4, at the Pediatric Academic Societies (PAS) annual meeting in Washington, DC.

"Texting while driving has become, in the words of Transportation Secretary Ray LaHood, a 'national epidemic,'" said principal investigator Alexandra Bailin, a research assistant at Cohen Children's Medical Center of New York.

Motor vehicle accidents are the leading cause of death among teenagers, and using a phone while driving significantly increases the risk of accidents in this age group. The specific act of texting while driving has been found to raise the risk of a crash by 23 times, leading many to conclude that texting while driving is more dangerous than driving while intoxicated.

"Although teens may be developmentally predisposed to engage in risk-taking behavior, reducing the prevalence of texting while driving is an obvious and important way to ensure the health and safety of teen drivers, their passengers and the surrounding public," Bailin said.

To determine the prevalence of texting while driving among youths, Bailin and her colleagues analyzed data from the 2011 Youth Risk Behavior Survey of 7,833 high school students who were old enough to get a driver's license in their state.

The Centers for Disease Control and Prevention conducts the national survey every two years to monitor six types of health-risk behaviors that contribute to the leading causes of death, disability and social problems among U.S. youths. For the first time, the 2011 survey included a question about texting while driving: "During the past 30 days, on how many days did you text or e-mail while driving a car or other vehicle?"

The researchers also sought to determine whether other high-risk behaviors are associated with texting while driving and if state laws prohibiting drivers from texting are effective among high school students.

Survey results showed that males were more likely to text while driving than females (46 percent vs. 40 percent), and the prevalence of texting increased with age (52 percent of those over 18 years; 46 percent of 17-year-olds; 33 percent of 16-year-olds; and 26 percent of 15-year-olds).

Furthermore, teens who reported texting while driving were more likely to engage in other risky behaviors such as driving under the influence of alcohol, having unprotected sex and using an indoor tanning device.

"By identifying associated high-risk behaviors such as these," Bailin said, "it is our hope that we can develop more effective mechanisms to reduce texting while driving."

The researchers also found that state laws banning texting while driving had little effect: 39 percent of teens reported texting in states where it is illegal vs. 44 percent of teens in states that have no restrictions.

"Although texting while driving was slightly less common in states that prohibit it, the reality is that millions of teens text while driving," said senior investigator Andrew Adesman, MD, FAAP, chief of Developmental and Behavioral Pediatrics at Cohen Children's Medical Center of New York. "Regrettably, our analysis suggests that state laws do not significantly reduce teen texting while driving.

"Technological solutions will likely need to be developed to significantly reduce the frequency of texting while driving," Dr. Adesman concluded. "When it comes to teen texting while driving, phones will have to get smarter if they are to protect teens (and others) from doing dumb things."

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The above story is reprinted from materials provided by American Academy of Pediatrics, via EurekAlert!, a service of AAAS.

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Disclaimer: This article is not intended to provide medical advice, diagnosis or treatment. Views expressed here do not necessarily reflect those of ScienceDaily or its staff.

Source: http://feeds.sciencedaily.com/~r/sciencedaily/top_news/top_health/~3/TQfvtgx1iaM/130504163308.htm

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Finally, Drones That Deliver Beer

We've seen tacocopters and burrito bombers, and though those will probably be real someday, neither is going to be really delivering anything any time soon. These beer drones, on the other hand, might actually be delivering real beer to real people. In South Africa. Maybe.

Read more...

Source: http://feeds.gawker.com/~r/gizmodo/full/~3/J0jnwi958vU/finally-drones-that-deliver-beer-491313956

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Study shows that individual brain cells track where we are and how we move

Friday, May 3, 2013

Leaving the house in the morning may seem simple, but with every move we make, our brains are working feverishly to create maps of the outside world that allow us to navigate and to remember where we are.

Take one step out the front door, and an individual brain cell fires. Pass by your rose bush on the way to the car, another specific neuron fires. And so it goes. Ultimately, the brain constructs its own pinpoint geographical chart that is far more precise than anything you'd find on Google Maps.

But just how neurons make these maps of space has fascinated scientists for decades. It is known that several types of stimuli influence the creation of neuronal maps, including visual cues in the physical environment ? that rose bush, for instance ? the body's innate knowledge of how fast it is moving, and other inputs, like smell. Yet the mechanisms by which groups of neurons combine these various stimuli to make precise maps are unknown.

To solve this puzzle, UCLA neurophysicists built a virtual-reality environment that allowed them to manipulate these cues while measuring the activity of map-making neurons in rats. Surprisingly, they found that when certain cues were removed, the neurons that typically fire each time a rat passes a fixed point or landmark in the real world instead began to compute the rat's relative position, firing, for example, each time the rodent walked five paces forward, then five paces back, regardless of landmarks. And many other mapping cells shut down altogether, suggesting that different sensory cues strongly influence these neurons.

Finally, the researchers found that in this virtual world, the rhythmic firing of neurons that normally speeds up or slows down depending on the rate at which an animal moves, was profoundly altered. The rats' brains maintained a single, steady rhythmic pattern.

The findings, reported in the May 2 online edition of the journal Science, provide further clues to how the brain learns and makes memories.

The mystery of how cells determine place

"Place cells" are individual neurons located in the brain's hippocampus that create maps by registering specific places in the outside environment. These cells are crucial for learning and memory. They are also known to play a role in such conditions as post-traumatic stress disorder and Alzheimer's disease when damaged.

For some 40 years, the thinking had been that the maps made by place cells were based primarily on visual landmarks in the environment, known as distal cues ? a tall tree, a building ? as well on motion, or gait, cues. But, as UCLA neurophysicist and senior study author Mayank Mehta points out, other cues are present in the real world: the smell of the local pizzeria, the sound of a nearby subway tunnel, the tactile feel of one's feet on a surface. These other cues, which Mehta likes to refer to as "stuff," were believed to have only a small influence on place cells.

Could it be that these different sensory modalities led place cells to create individual maps, wondered Mehta, a professor with joint appointments in the departments of neurology, physics and astronomy. And if so, do these individual maps cooperate with each other, or do they compete? No one really knew for sure.

Virtual reality reveals new clues

To investigate, Mehta and his colleagues needed to separate the distal and gait cues from all the other "stuff." They did this by crafting a virtual-reality maze for rats in which odors, sounds and all stimuli, except distal and gait cues, were removed. As video of a physical environment was projected around them, the rats, held by a harness, were placed on a ball that rotated as they moved. When they ran, the video would move along with them, giving the animals the illusion that they were navigating their way through an actual physical environment.

As a comparison, the researchers had the rats ? six altogether ? run a real-world maze that was visually identical to the virtual-reality version but that included the additional "stuff" cues. Using micro-electrodes 10 times thinner than a human hair, the team measured the activity of some 3,000 space-mapping neurons in the rats' brains as they completed both mazes.

What they found intrigued them. The elimination of the "stuff" cues in the virtual-reality maze had a huge effect: Fully half of the neurons being recorded became inactive, despite the fact that the distal and gate cues were similar in the virtual and real worlds. The results, Mehta said, show that these other sensory cues, once thought to play only a minor role in activating the brain, actually have a major influence on place cells.

And while in the real world, place cells responded to fixed, absolute positions, spiking at those same positions each time rats passed them, regardless of the direction they were moving ? a finding consistent with previous experiments ? this was not the case in the virtual-reality maze.

"In the virtual world," Mehta said, "we found that the neurons almost never did that. Instead, the neurons spiked at the same relative distance in the two directions as the rat moved back and forth. In other words, going back to the front door-to-car analogy, in a virtual world, the cell that fires five steps away from the door when leaving your home would not fire five steps away from the door upon your return. Instead, it would fire five steps away from the car when leaving the car. Thus, these cells are keeping track of the relative distance traveled rather than absolute position. This gives us evidence for the individual place cell's ability to represent relative distances."

Mehta thinks this is because neuronal maps are generated by three different categories of stimuli ? distal cues, gait and "stuff" ? and that all are competing for control of neural activity. This competition is what ultimately generates the "full" map of space.

"All the external stuff is fixed at the same absolute position and hence generates a representation of absolute space," he said. "But when all the stuff is removed, the profound contribution of gait is revealed, which enables neurons to compute relative distances traveled."

The researchers also made a new discovery about the brain's theta rhythm. It is known that place cells use the rhythmic firing of neurons to keep track of "brain time," the brain's internal clock. Normally, Mehta said, the theta rhythm becomes faster as subjects run faster, and slower as running speed decreases. This speed-dependent change in brain rhythm was thought to be crucial for generating the 'brain time' for place cells. But the team found that in the virtual world, the theta rhythm was uninfluenced by running speed.

"That was a surprising and fascinating discovery, because the 'brain time' of place cells was as precise in the virtual world as in the real world, even though the speed-dependence of the theta rhythm was abolished," Mehta said. "This gives us a new insight about how the brain keeps track of space-time."

The researchers found that the firing of place cells was very precise, down to one-hundredth of a second, "so fast that we humans cannot perceive it but neurons can," Mehta said. "We have found that this very precise spiking of neurons with respect to 'brain-time' is crucial for learning and making new memories."

Mehta said the results, taken together, provide insight into how distinct sensory cues both cooperate and compete to influence the intricate network of neuronal activity. Understanding how these cells function is key to understanding how the brain makes and retains memories, which are vulnerable to such disorders as Alzheimer's and PTSD.

"Ultimately, understanding how these intricate neuronal networks function is a key to developing therapies to prevent such disorders," he said.

In May, Mehta joined 100 other scientists in Washington, D.C., to help shape President Obama's BRAIN Initiative (Brain Research through Advancing Innovative Neurotechnologies), with the goal of trying to tease out how this most complicated of organs works.

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University of California - Los Angeles: http://www.newsroom.ucla.edu

Thanks to University of California - Los Angeles for this article.

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Source: http://www.labspaces.net/128120/Study_shows_that_individual_brain_cells_track_where_we_are_and_how_we_move

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Perform Pachelbel On a Pint-Sized Playable Pop-up Paper Piano

Taking those electronic music playing greeting cards to a whole new level, Antonella Nonnis created a playable paper piano that she eventually hopes to turn into one of the awesomest pop-up books you've ever seen.

The paper keys are all lined with a strip of copper foil that's wired to a series of megaohm resistors and an Arduino. Using the same capacitive approach as your smartphone's display, the electronics can sense when and which key is being pressed to produce the appropriate synthesized tone. So instead of only playing Happy Birthday, one day a card could be used to play an entire concerto. [Antonella Nonnis via Creative Applications]

Source: http://gizmodo.com/perform-pachelbel-on-a-pint-sized-playable-pop-up-paper-489071555

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Kobe Bryant battles mom, NJ business over auction

CAMDEN, N.J. (AP) ? Kobe Bryant is in a court battle to try to keep his mother from auctioning off mementoes from his high school days in Pennsylvania and his early NBA career.

A southern New Jersey auction house is suing for the right to sell the stuff after the NBA star's lawyers wrote the firm to say it could not.

Bryant contends that his mother, Pamela Bryant, doesn't have the right to sell the collectibles including his Lower Merion High School letters, a 2000 NBA championship ring and hundreds of other items.

Berlin, N.J.-based Goldin Auctions says Bryant told his mother years ago that he didn't want the items.

Legal papers say Pamela Bryant intended to use her $450,000 advance to buy a new home.

Associated Press

Source: http://hosted2.ap.org/APDEFAULT/347875155d53465d95cec892aeb06419/Article_2013-05-03-Kobe%20Bryant-Lawsuit/id-15eed8e2270244e095dccd44107c8fbb

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Peeping Toms fall through ceiling of women?s bathroom

No peeping (ThinkStockPhotos)

Two men fell through a woman?s restroom ceiling while attempting to spy on them, police said.

According to a story in the Atlanta Journal Constitution, the incident occurred at an Atlanta-area movie complex, the Venture Value Cinema in Duluth, Ga.

The suspects, 26-year-old Eduard Petrovich Kovynev and 27-year-old Eduard Alexander Kovynev, who apparently were more interested in the bathroom than the big screen, allegedly crawled through the men?s room ceiling over to the women?s room ?and then fell through, landing directly inside the stalls.?

According to the paper, an officer wrote in the arrest warrant that the suspects were "invading the privacy of various customers."

The two were charged with peeping Tom and criminal damage to property.

Most of the time, unwelcome ogling happens without the victims? knowledge. But in a case in Los Angeles, a woman discovered a peeping Tom had been taking cellphone pictures up her skirt thanks to another shopper telling her what happened and pointing out the suspect.

The quick thinking 27-year-old victim took photos of the suspect?s license plate and called the police. The peeping Tom's actions were caught on tape, and the suspect was arrested. According to a Los Angeles Police Department press release, ?In most cases, these crimes are never seen and rarely reported. Thankfully, another customer saw the man.?

Source: http://news.yahoo.com/blogs/sideshow/peeping-toms-fall-ceiling-women-bathroom-163654790.html

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Obama moving toward sending lethal arms to Syrian rebels, officials say (Washington Post)

Share With Friends: | | Politics - Top Stories News, RSS and RSS Feed via Feedzilla.

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Eternal flame at JFK grave undergoing repairs

MADRID, April 30 (Reuters) - Teams for Tuesday's Champions League semi-final second leg between Real Madrid and Borussia Dortmund at the Bernabeu: Real Madrid: 41-Diego Lopez; 15-Michael Essien, 4-Sergio Ramos, 2-Raphael Varane, 5-Fabio Coentrao; 19-Luka Modric, 14-Xabi Alonso; 22-Angel Di Maria, 10-Mesut Ozil, 7-Cristiano Ronaldo; 20-Gonzalo Higuain Borussia Dortmund: 1-Roman Weidenfeller; 26-Lukasz Piszczek, 4-Neven Subotic, 15-Mats Hummels, 29-Marcel Schmelzer; 8-Ilkay Guendogan, 6-Sven Bender; 16-Jakub Blaszczykowski, 10-Mario Goetze, 11-Marco Reus; 9-Robert Lewandowski Referee: Howard ...

Source: http://news.yahoo.com/eternal-flame-jfk-gravesite-undergoing-repairs-162604202.html

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